Search results for "GNAS complex locus"

showing 5 items of 5 documents

Evaluation of the Possibility to Detect Circulating Tumor DNA From Pituitary Adenoma

2019

Objective: Circulating free DNA (cfDNA) in general and circulating tumor DNA (ctDNA) in particular is becoming an increasingly used form of liquid biopsy biomarkers. In this study, we are investigating the ability to detect ctDNA from the plasma of pituitary adenoma (PA) patients. Design: Tumor tissue samples were obtained from planed PA resections, before which blood plasma samples were taken. Somatic variants found in PA tissue samples were evaluated in related cfDNA, isolated from plasma samples. Methods: Sanger sequencing, as well as previously obtained whole-exome sequencing data, were used to evaluate somatic variants composition in tumor tissue samples. cfDNA was isolated from the sa…

0301 basic medicineSomatic cellEndocrinology Diabetes and Metabolism030209 endocrinology & metabolismpituitary adenomaBiologylcsh:Diseases of the endocrine glands. Clinical endocrinologyDNA sequencing03 medical and health sciencessymbols.namesakeGNAS0302 clinical medicineEndocrinologyBlood plasmaTaqManGNAS complex locusLiquid biopsyOriginal ResearchSanger sequencingcirculating tumor DNAlcsh:RC648-665AmpliconMolecular biology030104 developmental biologybiology.proteinsymbolsnext-generation sequencingcompetitive allele-specific TaqManFrontiers in Endocrinology
researchProduct

Boy with pseudohypoparathyroidism type 1a caused byGNASgene mutation (deltaN377), Crouzon-like craniosynostosis, and severe trauma-induced bleeding

2009

We report on a 6-month-old boy with craniosynostosis, pseudohypoparathyroidism type 1a (PHP1A), and a GNAS gene mutation. He had synostoses of the coronal, frontal, and sagittal sutures, brachyturricephaly, and hydrocephaly. He also had congenital hypothyroidism, round face, full cheeks, shortness of limbs, mild developmental delay, and muscular hypotonia. Because of progressive hydrocephaly, the synostosis was corrected surgically but circulatory decompensation led to disseminated intravascular coagulation and cerebral infarctions. Our patient died 8 days later. Postmortem molecular studies of GNAS, the gene for guanine nucleotide-binding protein, alpha-stimulating activity polypeptide (ge…

Malemedicine.medical_specialtyPathologyCraniosynostosisFatal OutcomeInternal medicineChromograninsCongenital HypothyroidismGTP-Binding Protein alpha Subunits GsGeneticsmedicineGNAS complex locusHumansGenetic Predisposition to DiseaseGenetics (clinical)PseudohypoparathyroidismDisseminated intravascular coagulationbiologyMuscular hypotoniabusiness.industryCraniofacial DysostosisInfantDysostosisSynostosismedicine.diseaseCongenital hypothyroidismEndocrinologyBrain InjuriesPseudohypoparathyroidismMutationbiology.proteinbusinessIntracranial HemorrhagesHydrocephalusAmerican Journal of Medical Genetics Part A
researchProduct

A new heterozygous mutation (D196N) in the Gs alpha gene as a cause for pseudohypoparathyroidism type IA in a boy who had gallstones

2011

Background Pseudohypoparathyroidism (PHP) is characterized by hypocalcemia and hyperphosphatemia in association with an increased secretion of parathyroid hormone (PTH) due to decreased target tissue responsiveness to PTH. Patients with PHP type Ia are not only resistant to PTH, but also to other hormones that bind to receptors coupled to stimulatory G protein (Gsalpha). PHP Ia and Albright hereditary osteodystrophy (AHO) are caused by a reduced activity of the Gsalpha protein. Heterozygous inactivating Gs alpha (GNAS) gene mutations have been identified in these patients. Methods We studied a boy with PHP Ia. During follow-up the patient developed elevated liver enzyme serum levels and abd…

Malemusculoskeletal diseasesHeterozygotemedicine.medical_specialtyErythrocytesFoot Deformities CongenitalEndocrinology Diabetes and MetabolismMutation MissenseParathyroid hormoneGallstonesGene mutationHyperphosphatemiaEndocrinologyInternal medicineChromograninsGTP-Binding Protein alpha Subunits GsGNAS complex locusHumansMedicineMissense mutationnatural sciencesAmino Acid SequenceChildConserved SequencePseudohypoparathyroidismBase SequenceSequence Homology Amino Acidbiologybusiness.industryDNAExonsGallstonesmedicine.diseasePedigreeCholesterolEndocrinologyAmino Acid SubstitutionPseudohypoparathyroidismPediatrics Perinatology and Child Healthbiology.proteinbusinessHand Deformities CongenitalHormoneJournal of Pediatric Endocrinology and Metabolism
researchProduct

Whole exome sequencing and system biology analysis support the "two-hit" mechanism in the onset of Ameloblastoma

2021

Background Ameloblastoma is the most frequent odontogenic tumor. Various evidence has highlighted the role of somatic mutations, including recurrent mutation BRAF V600E, in the tumorigenesis of Ameloblastoma, but the intact genetic pathology remains unknown. Material and Methods We sequenced the whole exome of both tumor tissue and healthy bone tissue from four mandibular ameloblastoma patients. The identified somatic mutations were integrated into Weighted Gene Co-expression Network Analysis on publicly available expression data of odontoblast, ameloblast, and Ameloblastoma. Results We identified a total of 70 rare and severe somatic mutations. We found BRAF V600E on all four patients, sup…

Proto-Oncogene Proteins B-rafOdontogenic TumorsBiologymedicine.disease_causeAmeloblastomaGermline mutationOral Cancer and Potentially malignant disordersExome SequencingmedicineGNAS complex locusspainHumansMissense mutationrisk factorsawarenessAmeloblastomaBiologyGeneral DentistryExomeExome sequencingUNESCO:CIENCIAS MÉDICASResearchoral cancermedicine.diseaseOtorhinolaryngologyMutationsurveys and questionnairesCancer researchbiology.proteinSurgeryCLTCCarcinogenesis
researchProduct

Fertility in McCune Albright syndrome female: A case study focusing on AMH as a marker of ovarian dysfunction and a literature review.

2021

Abstract Background The molecular basis of McCune Albright syndrome (MAS) is a recurrent GNAS Postzygotic gain of function sporadic mutation, resulting in a mosaic disease. Most of girls present precocious puberty, caused by the development of recurrent ovarian cysts with autonomous Hyperestrogenic stimulation. After menarche, the majority of patients with ovarian GNAS mutation have menstrual disturbances and infertility. Objectives We wanted to focus on the fertility of MAS females and propose an appropriate management, by a detailed case report and an exhaustive review of the literature on fertility and pregnancy in MAS females. Results We present the case of a 29-year-old MAS female, who…

musculoskeletal diseasesInfertilityAdultAnti-Mullerian Hormoneendocrine systemmedicine.medical_specialtyendocrine system diseasesGenetic counselingmedicine.medical_treatmentOvariectomyFertilization in VitroFibrous Dysplasia PolyostoticMcCune–Albright syndrome03 medical and health sciences0302 clinical medicineFollicular phasemedicineGNAS complex locusPrecocious pubertyHumansGynecology030219 obstetrics & reproductive medicineIn vitro fertilisationbiologybusiness.industryObstetrics and GynecologyOophorectomymedicine.diseasefemale genital diseases and pregnancy complicationsReproductive Medicine030220 oncology & carcinogenesisbiology.proteinFemalebusinessInfertility Femalehormones hormone substitutes and hormone antagonistsJournal of gynecology obstetrics and human reproduction
researchProduct